Highlights
Assessment
You are required to complete the name and date fields on the first page of this document. Failure to do so will result in your submission counting towards an attempt and will not be assessed as it is incomplete.
Number of Attempts:
Should your 1st and 2nd attempt be deemed as Not Yet Satisfactory (NYS), your assessor will provide feedback informing you of the areas requiring additional or further information. You will be required to submit your updated attempt on a resubmission form template. If your 3rd attempt is deemed Not Yet Satisfactory (NYS), you will be required to schedule a call with an assessor to address the areas deemed Not Yet Satisfactory (NYS). If you fail to attend the scheduled call for discussion, you will receive an overall Not Yet Satisfactory (NYS) result for this assessment task.
General information:
Each unit will have theoretical and practical components. The practical components for this unit will be addressed within the clinical practice workshop which you are required to attend to finalise individual unit outcomes.Students are advised to read the Student Unit Guide prior to beginning assessment to ensure a clear understanding of the unit requirements and assessment tasks involved. Students must satisfactorily complete all associated assessment tasks to be deemed competent overall in this unit of competency.Students are permitted to use the Student Learner Guide and additional resources available to support responses, however where relevant it is expected that references are listed or identified for any work that is not your own words. Academic misconduct is monitored and plagiarism will be penalised as per the Academic Integrity Policy.
Medical Condition:
ACUTE CORONARY SYNDROME
In your own words, explain what Acute Coronary Syndrome (ACS) is?
Acute coronary syndrome is a term used to describe a range of conditions associated with sudden, reduced blood flow to the heart.One such condition is a heart attack (myocardial infarction) — when cell death results in damaged or destroyed heart tissue. Even when acute coronary syndrome causes no cell death, the reduced blood flow changes how your heart works and is a sign of a high risk of heart attack
2. Explain the pathophysiology of Acute Coronary Syndrome?
Plaque rupture has dominated our thinking about acute coronary syndromes (ACS) pathophysiology for decades. However, current evidence suggests that a sole focus on plaque rupture obscures other mechanisms that may mandate different management strategies. First, coronary artery thrombosis caused by plaque rupture can occur with or without signs of concomitant inflammation. This distinction may have substantial therapeutic implications as direct anti-inflammatory interventions for atherosclerosis are emerging. Coronary artery thrombosis caused by plaque erosion may be on the rise in an era of intense lipid lowering. Identification of patients with ACS resulting from erosion may permit a less invasive approach to management than the current standard of care; furthermore, new therapeutic targets are emerging as hyaluronic acid metabolism alterations seem to play a key role in this setting. Finally, ACS may occur without apparent epicardial coronary artery thrombus or stenosis. Such events may arise from spasm or coronary microvascular dysfunction. Emerging management strategies may likewise apply selectively to this category of ACS.
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