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Mary’s wound was swollen and painful, red and warm when touched and purulent. This appearance of the wound is due to the inflammatory response which is not supposed to be stopped with anti-inflammatory drugs (Lee, 2016). The bleeding wound after the injury releases ATP triphosphateand histamine from the injured tissues which attract, white blood cells to the wound area. Ends of blood vessels constrict to trigger a clotting cascade. The wound area begins to gel and a clot formation takes places, as platelets stick together. This may prevent wound infection. As the clotting takes place a mesh is formed by a protein fibrin, in WBCs (which blood cells) become trapped to form a forming a strong clot. Exposed clot surface dries to form a vasodilation. As a result of this blood supply is increased to the injury and space in the endothelium allows blood cells to enter into the interstitial spaces. This results in repair of the wound and clearing of the debris. The phagocytic process (engulfing and digesting bacteria and contaminants) begins as the Neutrophils invaded the wound site(Lee, 2016). During the late inflammatory stage, monocytes get transformed into macrophages. The macrophages (specialized WBCs) move through injured dermis, engulfing microbes and debris. Macrophages also secrete growth factors, vital for the healing process. From the epidermis, keratinocytes move to the open edges of the wound, in order to protect the deep exposed tissues. This stage is characterized by red, hot, swollen and painful wound. Inflammation is an important part of the healing and should not be subdued with inflammatory drugs (Marieb, 2016). Thus in Mary’s case a painful, warm, red and swollen wound is due to tissue injury which triggered an inflammatory response in the body. The dead cells and debris with infection due to microbes, made the wound purulent.