NURS2500 - Pathophysiology for Nurses - Medical Science Assignment Help

Assignment Task

Pathophysiology:

Psoriasis is known to be a prevalent skin disorder and a number of linked cellular alterations in the skin characterize this ailment (Araújo, Resende, Vieira, Fernandes & Brito, 2015). Pathophysiology of psoriasis is multi-factorial that includes abnormal differentiation of the epidermal keratinocytes, epidermal hyper proliferation, immunological changes and alterations in the skin and inflammation of the skin (Benjegerdes, Hyde, Kivelevitch & Mansouri, 2016). Hyperproliferation is described by accelerated DNA synthesis and a significantly reduced epidermis dropout rate. Abnormal keratinocyte separation entails elevated processing of many keratins (6 and 16) and changes in the production of several other keratins (1 and 10) that are usually generated in differentiated and affected skin. Inflammation tends to result from superficial and epidermis dermal layer infiltration of neutrophils and an infiltration of T lymphocytes in the dermis with the prevalence of CD8 + cells (Benjegerdes, Hyde, Kivelevitch & Mansouri, 2016).

Clinical Manifestations:

Cracked skin with dryness that sometimes results in bleeding, small scaling spots, red patches can be seen that are covered by the silvery thick spots, soreness or burning, itching, ridges nails or pitted and thickened nails and stiff and swollen joints (Köhm, Burkhardt & Behrens, 2015).

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